Low blood sugar levels which result from starvation can cause irreversible brain damage (Andersen; Integration). When starvation occurs, the brain has to use ketone bodies as a source of acetyl-CoA. It uses its might to maintain an electrical potential in the neurons so they delay to function. In starvation, neuronal signals from the brain induce the release of epinephrin from the adrenal medulla, the normal response to stress. This results in glycogen phosphorylate activation and inactivation of glycogen synthase, i.e. glucose storage is inhibited and its release into the blood stream promoted by the segmentation of glycogen stored in the liver. Glucagon secretion is generate and insulin secretion inhibited since insulin lowers the blood sugar level. Anaerobic breakdown of glycogen in skeletal muscle is also stimulated. go mobilization from adipose tissue is stimulated t
Medical Information. "Possible complications of eating disorders." 2005. 3 Sep. 2005.
everywhere the next few days, oxaloacetate is depleted in devising glucose and leave an accumulation of acetyl-CoA, which favors the formation of ketone bodies by the liver (Integration).
The brain uses these as fuel in place of glucose. When fat militia are depleted, the body begins breaking down essential proteins, which leads to loss of liver and heart function as these organs are broken down for fuel. Triacylglycerols would theoretically domiciliate enough zilch for the body for months, but would lead to morbidly high levels of ketone bodies.
hrough cAMP-mediated phosphorylation to provide ketone bodies for acetyl-CoA for the starving brain. Brain starvation can be rectify by returning food inlet to normal (Medical). However, during a period of starvation, the gastrointestinal tract shrinks in size and cannot keep the same amount of food as before. Food intake must be increased slowly,
Order your essay at Orderessay and get a 100% original and high-quality custom paper within the required time frame.
No comments:
Post a Comment